Pre-Congress Course

Osseous Orthodontics without flap elevation to accelerate orthodontic tooth movement
Young-Guk Park, S. Korea

The usual rate of tooth movement by conventional protocols of mechanotherapy signifies approximately 1 mm/month. The representative noxious complication over time involves the decay of compliance from the patients and consequent fatigue of the orthodontists.
A surgically induced transient osteopenia (RAP; regional acceleratory phenomenon) is perpetuated by tooth movement at the rate of 1 mm/week. The osteopenic milieu is clinically expedient with sound biologic foundation, and renders the orthodontic outcome more stable and less prone to complications.
Orthodontic tooth movement results from forces applied to tooth, that evoke cellular responses in the teeth and their surrounding tissues, including the periodontal ligament (PDL), alveolar bone, and gingiva. It is advantageous for the orthodontist to know the details of the biological events that unfold during tooth movement, because some of these details may differ from one remedy to another, due to variables such as gender, age, psychological status, nutritional habits, and drug consumption. The purpose of this presentation is, therefore, to emphasize the fact that orthodontics is a field of endeavor where mechanics and biology are integrated, and to stress the reality that tooth movement is conducted in individual human beings, each composed of a unique and intricate physiological system. Biological variations may be the foundation of the differences that are frequently observed in the outcomes of orthodontic treatment between patients with similar malocclusions, treated with different regimen.
This presentation will present evidence that minor surgical procedure by orthodontist oneself can receive accelerated rates of tooth movement with impunity. It will introduce the novel way to orthodontics as a system that enhances the rate of bony and periodontal response, thereby shortening the duration of treatment, and preventing long-lasting tissue damage.

CURRICULUM VITAE: Young-Guk Park

1. Educational Background
DMD, Kyung Hee University 1981
MSD, Kyung Hee University (Orthodontics) 1984
PhD, Kyung Hee University(Orthodontics and Oral Biology) 1990
MBA, Kyung Hee University (Healthcare Management) 2000
2. Academic Position
1990 to 2000
Assistant and Associate Professor of Orthodontics, Kyung Hee University
1994-1995
Visiting Assistant Professor, Harvard School of Dental Medicine
2000-to date
Professor and Chair, Department of Orthodontics, Kyung Hee University
2000-to date
Guest Professor, Osaka Dental University, Japan
3. Social Activities
Vice President, Korean Association of Orthodontists
Chief director, Korean Board of Dental Specialties Examination, Korean Dental Association
A member, Education Committee, FDI World Dental Federation
4. Publications in recent 3 years
Park YG et al; cAMP-PKA signaling pathway regulates bone resorption mediated by processing of cathepsin K in cultured mouse osteoclasts, Int Immunopharmacol 6(3): 947-956 (2006)
Park YG et al: PGE2 induces the gene expression of bone matrix metalloproteinase-1 in mouse osteoblasts by cAMP-PKA signaling pathway. Int J Biochem Cell Biol 37(2): 375-385 (2005)
Park YG et al: Effects of TGF-beta, TNF-alpha, IL-beta and IL-6 alone or in combination, and tyrosine kinase inhibitor on cyclooxygenase expression, prostaglandin E2 production and bone resorption in mouse calvarial bone cells, . Int J Biochem Cell Biol 36(11):2270-2280 (2004)
Park YG et al: Protective and anti-arthritic effects of deer antler aqua-acupuncture (DAA), inhibiting dihydroorotate dehydrogenase, on phosphate ions-mediated chondrocyte apoptosis and rat collagen-induced arthritis. Int Immunopharmacol 4(7):963-973 (2004)
Park YG et al: PGE2 induces IL-1beta gene expression in mouse osteoblasts through a cAMP-PKA signaling pathway Int Immunopharmacol 4(6):779-789 (2004)

 
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